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Oxidized LDL is a big gamer in a very severe sort of liver problem called non-alcoholic steatohepatitis (NASH for brief). Steatosis suggests fat accumulation and hepatitis suggests liver swelling, so if you put it all together, you get an inflammatory disease of fat build-up in the liver. You can check out this here if you want all the details, but the brief version is: fat build-up and persistent swelling in the liver are actually not fun. This paper and this one make the case for oxidized LDL as a major contributing aspect to liver inflammation in NASH. Generally, oxidized LDL damages a kind of liver cells called Kupffer cells.

However in the presence of too much oxidized LDL cholesterol, they get bloated or "foamy." Bloated or foamy Kupffer cells are pro-inflammatory and develop an inflammatory state in the liver generally, which contributes to NASH.

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Beta cells are the cells in your kidneys that make insulin. Insulin is the hormonal agent that you need amongst other things to correctly digest and metabolize carbs. If your beta cells aren't working, your insulin metabolism isn't working, which makes it impossible to digest carbs, manage blood sugar, and do all that good stuff. Damage to the beta cells is a feature of both type 1 and type 2 diabetes (in different methods and for various factors, however it's there in both). This paper went over the ways that oxidized LDL cholesterol damages the beta cells. Basically, oxidized  This Is Noteworthy  to the endoplasmic reticulum (ER for short), which belongs of every cell in your body.

Fewer beta cells = worse metabolic health, so by eliminating off the beta cells, oxidized LDL damages your metabolic health. Basically, no matter what part of your body you're looking at, oxidized LDL is bad news. And we have not even gotten to plaque in the arteries yet! However the point here is that even individuals who don't have much reason to fret about arterial plaque or cardiac arrest still have a great deal of factors to avoid oxidized LDL.